新陳代謝籠架設計用來裝載15個獨立的MMC100小鼠新陳代謝籠。獨特的設計可以將每一個代謝籠方便的安裝和取下。設計提供了籠子的穩(wěn)定支撐，而不需要再增加其他的復雜的固定裝置或鉗位設計。只需簡單將籠子插入到孔中即可。

1  設計容納多達15個代謝籠
2  容易組裝提供的硬件
3  全鋁合金建造
4  耐用的烤粉環(huán)氧樹脂
5  鋁結(jié)構(gòu)管腳
6  防滑水平調(diào)節(jié)
7  鍍鋅的硬件
8  可選的不銹鋼硬件
9  可選的帶旋轉(zhuǎn)鎖和車輪制動器的旋轉(zhuǎn)腳輪

1  設計容納多達15個代謝籠
2  容易組裝提供的硬件
3  全鋁合金建造
4  耐用的烤粉環(huán)氧樹脂
5  鋁結(jié)構(gòu)管腳
6  防滑水平調(diào)節(jié)
7  鍍鋅的硬件
8  可選的不銹鋼硬件
9  可選的帶旋轉(zhuǎn)鎖和車輪制動器的旋轉(zhuǎn)腳輪

新陳代謝籠架設計用來裝載15個獨立的MMC100小鼠新陳代謝籠。獨特的設計可以將每一個代謝籠方便的安裝和取下。設計提供了籠子的穩(wěn)定支撐，而不需要再增加其他的復雜的固定裝置或鉗位設計。只需簡單將籠子插入到孔中即可。

KynurenineInfusion Disrupts Normal Blood Pressure Circadian Rhythms

DebraL IrsikW.BollagC.Isales

Biology, Medicine

The FASEB Journal   1 May 2022

The data suggest that kynurenine treatment impacts renalfunction and may contribute to the development of CKD, a significant riskfactor for cardiovascular disease, even in the context of normotension.

Changes inOxygen Consumption and Metabolomic Profiles in the Kidney of Sprague‐Dawley Rat fed a High‐Salt Diet

S.ShimadaBrianR. HoffmannAndrewS. GreeneMingyuLiangR.DashA.Cowley

Medicine, Biology

The FASEB Journal  1May 2022

It is concluded that a high‐salt diet increases oxygenconsumption and alters the metabolomic profiles of the kidney.

Toward the InterdisciplinaryTheory and Research

O.Yanitsky

Creative Education  6 March 2020

The article represents some thoughts and considerationson an interdisciplinary theory and research based on the relevant scientificworks and the author’s personal and “big family” experience and…

Toward theInterdisciplinary Theory and Research

AixaHafsha  19 April 2020

The article represents some thoughts and considerationson an interdisciplinary theory and research based on the relevant scientificworks and the author’s personal and “big family” experience and… 

High Salt DietIncreases Renal Oxygen Consumption in Sprague‐Dawley Rats

S.ShimadaR.DashA.Cowley

The FASEB Journal  1May 2021

It appears that in normal SD rats, the increased tubular metabolic workassociated with a high salt diet is initially accommodated by increasedperfusion (RBF) and a gradual increase of O2 consumption while extractionremains unchanged for a two‐week period.

Chronic RenalArtery Insulin Infusion Decreases 24 hr/day Renal Blood Flow in Diabetic Rats

M.BrandsW.Bush   Medicine, Biology

The FASEB Journal  1May 2021

A chronic renal vasoconstrictor influence of insulinunder diabetic conditions is suggested, which suggests that the decrease ininsulin may contribute directly to the renal vasodilation that occurs in type 1diabetes.

M.CrostelliO.MazzaM.MarianiD.MascelloC.Iorio

International Orthopaedics 14 September 2018

Bracing is a viable treatment strategy in thoracic lumbarkyphosis and can obtain good clinical results at medium terms follow-up even ifkyPHosis deformity remains in radiographs, as even smallest devices availableare often too big.

Precise Controlof Target Temperature Using N6-Cyclohexyladenosine and Real-Time Control ofSurface Temperature.

B.LaughlinI.BaileyS.RiceZ.BaratiLoriK. BogrenK.Drew

Therapeutic Hypothermia and Temperature…1 June 2018

An intravenous delivery protocol for CHA at 0.25mg/(kg·h) that, when coupled with conductive cooling, achievesand maintains a prescribed and consistent target Tb between 32°Cand 34°C for 24 hours.

Physiologicalhyperinsulinemia caused by acute hyperglycemia minimizes renal sodium loss bydirect action on kidneys.

DebraL IrsikM.Brands

Medicine, Biology American Journal of Physiology.Regulatory…  23 May 2018

The hypothesis that meal-induced increases in plasmainsulin are a major component of normal sodium homeostasis, and that thisoccurs by direct action of insulin on the kidney is supported.

The normalincrease in insulin after a meal may be required to prevent postprandial renalsodium and volume losses.

DebraL IrsikB.Blazer-YostA.StaruschenkoM.Brands

Medicine, Biology

American Journal of Physiology. Regulatory…   1 June 2017

It is demonstrated that the normal increase incirculating insulin in response to hyperglycemia may be required to preventexcessive renal sodium and volume losses and suggested that insulin may be a physiologicalregulator of sodium balance.